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Pitting edema in leg of patient with chr

heart failure (heart failure)

DHeart failure is expressed in a number of symptoms and does not in itself represent a clinical picture. It must therefore be researched which basic disease  is the cause of heart failure. First of all, you have to distinguish between right heart failure and left heart failure. If the blood backs up into the venous system of the body due to right heart failure, edema (swelling) occurs. These initially affect the legs and lower legs and, in the case of advanced heart failure, the upper parts of the body such as the back, liver or abdominal cavity. In the picture you can clearly see that dents appear in the fabric after you press your finger. If the pressure doesn't make dents, it's a case of lymphedema, which is caused by the lymph vessels and not the heart.

In left heart failure, the blood accumulates in front of the left heart and thus in the lungs. As a result, water is excreted into the alveoli and pulmonary edema occurs. There are early signs of left ventricular failure that are used by the secret service. In the 1990s, for example, I heard that CIA agents were examining the speeches of politicians in the aging Soviet parliament for weaknesses. The agents watched how many words a politician could speak before he had to catch his breath again. If it was only a few words, it spoke for left heart failure and the CIA estimated the life expectancy of the politicians in this way.

Shortness of breath occurs as part of cardiac insufficiency, initially only during exertion and later also at rest. With progressive cardiac insufficiency, the described edema also occurs.

The causes are manifold:

Coronary heart disease,heart muscle inflammation,Dilated cardiomyopathy, high blood pressure, heart valve defects, diabetes mellitus and radiation or chemotherapy drugs in cancer treatment. In general, heart failure often creeps in in older people. In this context we speak of the old age heart. Such a correct diagnosis cannot be made with the old heart. It just works worse than a young heart, resulting in poor pumping.


The diagnosis can often be made on the basis of the anamnesis and the listening. The weak pumping is easily visible in the ultrasound. The NT per BNP value is available to us as a laboratory parameter. This is increased in heart failure. The laboratory value and the ultrasound examination are reliable options for therapy monitoring.


Conventional medicine mainly uses ACE inhibitors, AT1 blockers, diuretics and sometimes also beta blockers. These measures also have an effect. But this is often not enough. Therefore, the therapy can be extended by naturopathic medicines such as strophanthin and hawthorn (Crategutt). A heart failure is always a mitochondrial weakness. The mitochondria must constantly provide the energy in the form of ATP for the heartbeat. Therefore, mitochondria-strengthening substances such as magnesium, coenzyme Q 10 and NADH support the heat output. The breakthrough is often only achieved through cell-specific therapy. These are cell substances from the young animal, which can regenerate the heart cell due to their information content and as a repair substance.

I have an impressive example:

An 82-year-old patient had a serious heart condition and was being treated in the hospital according to all the rules of the art when she appeared in our health clinic.  She was discharged in such a weak condition that it was impossible for her to sit up to take care of at home alone. That's why she decided on a private spa stay. I myself believed that she would not survive the next 14 days and therefore telephoned the relatives. She walked on the rollator and had shortness of breath at rest. I punctured her pleural effusions (water accumulations under the lungs), but that didn't help much because they quickly formed again. I injected her with cell components from the lamb and she recovered within 2 days. Her NT-Pro BNP value as a measure of cardiac insufficiency dropped from 45637 to 2714, standard value: <589. The value was still elevated, but the patient could hardly feel the slight cardiac insufficiency that was still present. She could walk without a walker and took part in social life again. The sleep disturbances disappeared. I stayed in contact with her longer and found out that she was even working in the garden again.

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